Showing posts with label genetics. Show all posts
Showing posts with label genetics. Show all posts

Tuesday, December 13, 2011

Muddiest Point

My muddiest point is when you are doing pedigrees, is it always contain X and/or Y to show male or female or is it always just dominant and recessive letters, example B-brown b-blonde? Or can it be either or depending on the question?

Muddiest Point

my muddiest points are the meaning of autosomal and sex-linked. I know that they are opposites (autosomal is the opposite of sex- linked), but what do they really mean?

AndyD's Muddiest Point

My muddiest point is the difference between autosomal recessive and autosomal dominant and how they work.

Muddiest Point

My muddiest point is how do you know if a problem is a certain type of autosomal or if it is a sex linked problem.

Monday, December 12, 2011

Muddiest Point

Can you please explain to me how cells can be transformed?

Muddiest Point

Can you please explain to me how cells can be transformed?

My Muddiest Point

My muddiest point in this unit was the differences between prophase 1 and prophase 2 of the two phases of meiosis. How are they different?

Muddiest Point

My muddiest point is the difference between autosomal recessive and autosomal dominant what is the difference. How do they work.

Sunday, December 11, 2011

Muddiest Point 11/5

My muddiest point are the haploid and diploid. I don't really understand what they are.

Nuerofibromatosis

Neurofibromatosis or NF1 is the growth of noncancerous tumors that form underneath the skin. This disorder is caused by a mutation on chromosome number 17. This disorder is Autosomal Dominant and if one parent has the mutation then half the children will have NF1. When people get this dicease they develop Neurofibrons which are tumors that can block arteries and can cause a stroke or heart attack. Each persons symptoms vary because the genes are so different that the severity of the symptoms vary.

Doctors have not found a cure for NF1 but tumors can be surgically removed and corect malformed bones. In very few cases the tumors actually become cancerous which is then treated with chemotherapy. The wierd thing about NF1 is that they may only have a couple of tumors or they may have thousands.

Muddiest Point- PatrykS

My muddiest point is the autosomal recessive and autosomal dominant, how does that all work?

Muddiest Point 11/5

One of my muddiest points is the difference haploid and diploid. I am confused with the 2N and N stuff. Another muddiest point are the pedigrees. I don't really know how to read them or decide the genotypes, or whatever you have to do them. Is there an easier way to understand them?

Wilson Disease-Patryk Swietek

Wilson Disease
Wilson disease is a genetic disorder that doesn't let your body get rid of extra copper. Copper is required for your body but having too much of it is poisonous. To get Wilson disease, you need to inherit two not normal copies of ATP7B gene from each parent. To carry the disease, you only need one. The symptoms of this disease are attacks at the liver, the nervous system or both. This results in swelling of the liver, fluid buildup and being bruised more easily. Good news is that Wilson disease can be diagnosed with physical examination and laboratory tests. It can also be treated although it requires lifelong treatment to control copper amount in body. People who get the disorder detected early get good treatment, can have good health.


ATP7B GENE

The
ATP7B gene is pointed out in the image above. This gene is located on the long arm of chromosome 13.




Thursday, December 8, 2011

GabbyM's Muddiest Point

My muddiest point for this chapter is what kind of errors can happen during meiosis.

Tuesday, December 6, 2011

AndyD Scribe Class 12-6-11

Today in class we learned about Genetic Disorders!
We started out by BrooklynC stamping our homework stamp-sheet and then discussing the homework with our groups of 3. After everything was settled at the beginning of class we went over the homework as a class and went over the problems that were unclear to us. After the class discussion we took some notes on Probability, What autosomal was, and what sex linked was. Probability was one of the things we discussed as we went over our Pedigree homework. We discussed the chances of having male and female babies and babies with disorders.
We also made the probability ratio of males and females in our class and their siblings
(pictured below). Males on bottom females on top because for some reason the image turned out awkward.
















Homework
  • Up 51-52
  • Read Chapter 14.2 in textbook.
  • Quiz on Thursday on the Punnet Square w/ co-dominance and etc.
Next Scribe will be- MichaelM

Andy D Scribe Post 12-5-11.

Today in class we learned about PEDIGREES!
In class we first started out by discussing our homework with our new seating chart and groups that Mrs. Stein arranged for us. After our discussion with our peers we went over the correct answers as a class and discussed any of the questions that were kind of weirdly worded and tricky. After going through the answers we got into more detail with Blood typing and sex linked problems, especially the hybrid blood types.
Please ignore the names mentioned in the Pedigree above..... However the main lesson of today was Pedigrees. What are Pedigrees? They a chart made up of family members from one generation continuing on till the current. Pedigrees are used to trace disease or a specific trait within a family. Males in the chart are shown as Squares, and Females are shown as circles. However people that are shaded in have that trait or disease the is being tracked, women however with a half shaded circle are carriers of the disease or trait and may pass it on to one of their children and so on.

HOMEWORK!
  • UP 47-50
  • Read Chapter 14.1 in the textbook ( no duh where else?)
  • Quiz on how to solve all of the punnet squares and problems on how to find a genotype and phenotype ( codominance etc.), Pedigrees will also be on the quiz!

Monday, November 28, 2011

11/28

In class on 11-28-11. We started a NEW UNIT!! The new unit we are starting is about Meiosis, Genetics, and Genetic Technology. The way we started the new unit out was to compare and contrast Meiosis to Mitosis or review the phases of Meiosis (this was homework over thanksgiving break).




Today in class we took notes on Meiosis. Mrs. Stein asked us a question she said What is the goal of Meiosis? The goal of Meiosis is making the egg and sperm cell.



There are 23 pairs of homologous cells. In total there are 46. Each pair comes from your parents. Cells with a double set of chromosome are diploid (2n). Cells with one set of chromosomes are haploid (n). In Meiosis the chromosomes are haploiod. A haploid has half the number of chromosomes.



Where does Meiosis occur? Meiosis occurs in the ovary and testes.



Our body cells are diploid cells (2N) 2N=46 N=23


Gametes are sex cells and they are haploid. The fertilization restores the diploid state (N+N=2N).
Zygote is a fertilized egg (2N).

This is a picture of Meiosis I and Meiosis II.
In Meiosis I there is Prophase I, Metaphase I, Anaphase I and Telophase I. Then in Meiosis II the cells is formed a clevlege. The end result is Cytoskinesis and 4 cells are created. In this picture there is 2 cell divisions-Meiosis I and II.

The homework for today 11-28-11 was to read 11.4 with double bubble map or flow map (your choice).
Unit Packet 13-14-READ/COLOR/ANOTATE.

Next scribe is JessicaB :).

Thursday, November 17, 2011

EXTRA CREDIT!

Alright guys... 2 extra credit opportunities!

Click on this link and answer the following questions.
1. What color is the RNA backbone in this activity?
2. What 3 amino acids make up the polypeptide chain at the end of the protein synthesis activity?
First 4 people to email me the answers will get extra credit!


Opportunity #2... click on this link, enter in your FULL name... and complete the 'quiz'. You will get extra credit based on your score. This opportunity will expire on Sunday at midnight!